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Advanced MASH-related liver fibrosis is typically the result of a chronic, sustained inflammatory response involving multiple factors, characterized by abnormal deposition of type I collagen in the extracellular matrix (ECM), with the activation of HSCs being a key driver of liver fibrosis (Chan et al

Author contributions SM: Conceptualization, Writing original draft

doi: 10.1007/BF00294431 383 PlioplysAVPlioplysS

250,251,252 UA induced inflammation and relative mechanism Uric acid signaling triggers the activation of several transcription factors, such as NF-B or AP-1, through the activation of MAPK p38 and ERK pathways, resulting in the production of reactive oxygen species (ROS) under the conditions of hyperuricemia