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It also found that the increased post-synaptic mechanism was due to the involvement of -amino-3-hydroxyl-5-methyl-4-isoxazolepropionate (AMPA) receptors

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Based on these findings, we speculate that ROS production triggered by an HSV-1-directed metabolic switch and the interruption of GSH biosynthesis act together to promote ferroptosis in HSV-1-infected neural cells

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